Osteodystrophy in Children with Urolithiasis Caused by Primary Hyperparathyroidism

Authors

  • Abdusattor Akhatovich Nasirov epartment of Faculty Pediatric Surgery, Tashkent Medical Pediatric Institute, Tashkent, Uzbekistan
  • Fatkhillo Fayzievich Bayakhmedov epartment of Faculty Pediatric Surgery, Tashkent Medical Pediatric Institute, Tashkent, Uzbekistan
  • Timur Turaevich Norbayev Department of Faculty Pediatric Surgery, Tashkent Medical Pediatric Institute, Tashkent, Uzbekistan
  • Mukhamadjon Abdurakhmonovich Norov Tashkent Medical Pediatric Institute, Tashkent, Uzbekistan
  • Sultanmurad Bakhtiyorovich Abdurakhmanov Tashkent Medical Pediatric Institute, Tashkent, Uzbekistan

DOI:

https://doi.org/10.48112/acmr.v2i4.24

Keywords:

Urolithiasis, Primary Hyperparathyroidism, Kidneys, Alkaline Phosphatase, Pediatric Urology

Abstract

Abstract Views: 26

Carried out the retrospective analysis 52 children, ill nefrolitiasis of etiology hyperparathyroidism. By purpose showed up to study degree demineralization bony cloth as to forms primary hyperparathyroidism at children. The studies of the activity of alkalized phosphatase and ehoosteometrii will lend possibility to appreciate degree demineralization bony cloth and accordingly diferation PGPT as to forms and the weights of disease. Hyperparathyroidism contributes development demineralization the bony cloth of various intensity which is connected by physical charge on bonus cloth. Indicators characterizing bony changes in stone disease of etiology hyperparathyroidism were more at children in miscellaneous form PGPT and accordingly high indicators are noted at children with the disorder of the function of buds and had direct relationship with quantity and size of formed stone and osteoporosis of bones.

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Published

2021-12-31

How to Cite

Nasirov, A. A., Bayakhmedov, F. F., Norbayev, T. T., Norov, M. A., & Abdurakhmanov, S. B. (2021). Osteodystrophy in Children with Urolithiasis Caused by Primary Hyperparathyroidism. Advances in Clinical Medical Research, 2(4), 08–10. https://doi.org/10.48112/acmr.v2i4.24

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